Search results for "Alveolar cells"

showing 7 items of 7 documents

Basement Membrane Mimics of Biofunctionalized Nanofibers for a Bipolar-Cultured Human Primary Alveolar-Capillary Barrier Model

2017

In vitro reconstruction of an alveolar barrier for modeling normal lung functions and pathological events serve as reproducible, high-throughput pharmaceutical platforms for drug discovery, diagnosis, and regenerative medicine. Despite much effort, the reconstruction of organ-level alveolar barrier functions has failed due to the lack of structural similarity to the natural basement membrane, functionalization with specific ligands for alveolar cell function, the use of primary cells and biodegradability. Here we report a bipolar cultured alveolar-capillary barrier model of human primary cells supported by a basement membrane mimics of fully synthetic bifunctional nanofibers. One-step elect…

0301 basic medicinePolymers and PlasticsPolyestersNanofibersBiocompatible MaterialsBioengineeringNanotechnology02 engineering and technologyRegenerative medicineBasement MembranePermeabilityPolyethylene GlycolsBiomaterialsAlveolar cells03 medical and health sciencesTissue engineeringCell Line TumorCell AdhesionHuman Umbilical Vein Endothelial CellsMaterials ChemistrymedicineHumansBasement membraneTissue EngineeringTissue ScaffoldsChemistryEndothelial Cellsrespiratory system021001 nanoscience & nanotechnologyElectrospinningPolyester030104 developmental biologymedicine.anatomical_structureNanofiberBiophysicsSurface modification0210 nano-technologyBiomacromolecules
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Upregulation of activin-B and follistatin in pulmonary fibrosis: a translational study using human biopsies and a specific inhibitor in mouse fibrosi…

2014

Background: Activins are members of the TGF-ß superfamily of growth factors. First, we identified by expression array screening that activin-B and follistatin are upregulated in human idiopathic pulmonary fibrosis (IPF). Next, we wanted to clarify their specific role in lung fibrosis formation. Methods: We used specific antibodies for activin-A and -B subunits and follistatin to measure and localize their levels in idiopathic pulmonary fibrosis and control lung biopsies. To inhibit activin signaling, we used soluble activin type IIB receptor fused to the Fc portion of human IgG1 (sActRIIB-Fc) in two different mouse models of pulmonary fibrosis. Results: Activin-B and follistatin mRNA levels…

MalePathologyFollistatinPulmonary FibrosisPROTEINCell CountQuadriceps MuscleACTIVATIONIdiopathic pulmonary fibrosisMiceBMP-7FibrosisPulmonary fibrosisfollistatinInhibin-beta SubunitsGREMLINImmunity Cellularmedicine.diagnostic_testbiologyactivinsPIRFENIDONEPirfenidonerespiratory systemidiopathic pulmonary fibrosisMouse fibrosis model3. Good healthUp-RegulationActivinsmedicine.anatomical_structureACUTE EXACERBATIONmouse fibrosis modelembryonic structuresGROWTHBronchoalveolar Lavage Fluidhormones hormone substitutes and hormone antagonistsmedicine.drugResearch ArticleSignal TransductionPulmonary and Respiratory MedicineEXPRESSIONmedicine.medical_specialtyendocrine systemRecombinant Fusion ProteinseducationIdiopathic pulmonary fibrosisRespiratory MucosaAlveolar cellsINFLAMMATIONmedicineAnimalsHumansRNA MessengerLungbusiness.industrymedicine.diseaserespiratory tract diseasesMice Inbred C57BLPulmonary AlveoliDisease Models AnimalBronchoalveolar lavageProtein Biosynthesis3121 General medicine internal medicine and other clinical medicinebiology.proteinbusinessFollistatin
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GSE4-loaded nanoparticles a potential therapy for lung fibrosis that enhances pneumocyte growth, reduces apoptosis and DNA damage.

2021

© 2021 The Authors.

0301 basic medicineTelomeraseDNA damageApoptosismacromolecular substancesBleomycintelomeraseBiochemistryPulmonary fibrosisAlveolar cellsAlveolar cells03 medical and health scienceschemistry.chemical_compoundIdiopathic pulmonary fibrosisBleomycin0302 clinical medicineFibrosisPulmonary fibrosisGeneticsmedicineHumansMolecular BiologyTelomeraseLungLungNanopartículespulmonary fibrosisChemistrytechnology industry and agricultureFibrosi pulmonaralveolar cellsrespiratory systemmedicine.diseaseOxidative Stress030104 developmental biologymedicine.anatomical_structureAlveolar Epithelial CellsCancer researchGSE4NanoparticlesCollagenPeptides030217 neurology & neurosurgeryBiotechnologyDNA DamageFASEB journal : official publication of the Federation of American Societies for Experimental BiologyREFERENCES
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Morphogenetic (Mucin Expression) as Well as Potential Anti-Corona Viral Activity of the Marine Secondary Metabolite Polyphosphate on A549 Cells

2020

The mucus layer of the nasopharynx and bronchial epithelium has a barrier function against inhaled pathogens such as the coronavirus SARS-CoV-2. We recently found that inorganic polyphosphate (polyP), a physiological, metabolic energy (ATP)-providing polymer released from blood platelets, blocks the binding of the receptor binding domain (RBD) to the cellular ACE2 receptor in vitro. PolyP is a marine natural product and is abundantly present in marine bacteria. Now, we have approached the in vivo situation by studying the effect of polyP on the human alveolar basal epithelial A549 cells in a mucus-like mucin environment. These cells express mucins as well as the ectoenzymes alkaline phospha…

Aquatic OrganismsSecondary MetabolismVirus AttachmentPharmaceutical ScienceAdenylate kinaseRespiratory MucosaMucin 5ACdigestive systemArticle03 medical and health sciences0302 clinical medicinemucinPolyphosphatesDrug Discoveryotorhinolaryngologic diseasesExtracellularHumansReceptorlcsh:QH301-705.5neoplasmsinnate immunityPharmacology Toxicology and Pharmaceutics (miscellaneous)MUC1030304 developmental biologyA549 cellBiological Products0303 health sciencesBacteriaSARS-CoV-2ChemistryCell growthMucin-1MucinCOVID-19polyphosphaterespiratory systemImmunity Innatedigestive system diseasesSARS-CoV-2 spike proteinADKCell biologyATPlcsh:Biology (General)A549 Cells030220 oncology & carcinogenesishydrogelhuman alveolar cellsMarine Drugs
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Tachykinin-, calcitonin gene-related peptide-, and protein gene product 9.5-immunoreactive nerve fibers in alveolar walls of mammals.

1991

The presence and distribution of the presumed pan-neural marker protein gene product 9.5 (PGP)- and peptide-immunoreactive (ir) nerve fibers in alveolar walls of various species was investigated by light microscopic single and double staining immunohistochemistry. PGP-, tachykinin (TK)-, and calcitonin gene-related peptide (CRGP)-ir fibers were sparsely distributed in a similar pattern in alveolar walls of all species investigated. No vasoactive intestinal peptide-, peptide histidine isoleucine-, galanin-, and opioid-ir nerve fibers could be detected. PGP-ir fibers outnumbered those staining for TKs and CGRP. There was partial coexistence of PGP and TK as well as of TK and CRGP. PGP-, TK-, …

medicine.medical_specialtyAlveolar EpitheliumCalcitonin Gene-Related PeptideVasoactive intestinal peptideGuinea PigsCalcitonin gene-related peptideBiologyAlveolar cellsDogsNerve FibersSpecies SpecificityInternal medicineCricetinaeTachykininsmedicineAnimalsGalaninMammalsintegumentary systemMesocricetusGeneral NeuroscienceNeuropeptidesrespiratory systemMolecular biologyRatsPulmonary Alveolimedicine.anatomical_structureEndocrinologyCalcitoninPeripheral nervous systemCatsPulmonary alveolusUbiquitin ThiolesteraseBiomarkersNeuroscience letters
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Cytokeratins in normal lung and lung carcinomas

1984

The various epithelial cells of the lower respiratory tract and the carcinomas derived from them differ markedly in their differentiation characteristics. Using immunofluorescence microscopy and two-dimensional gel electrophoresis of cytoskeletal proteins from microdissected tissues we have considered whether cytokeratin polypeptides can serve as markers of cell differentiation in epithelia from various parts of the human and bovine lower respiratory tract. In addition , we have compared these protein patterns with those found in the two commonest types of human lung carcinoma and in several cultured lung carcinoma cell lines. By immunofluorescence microscopy, broad spectrum antibodies to c…

Pathologymedicine.medical_specialtyLung NeoplasmsCellular differentiationAdenocarcinomaBiologyCell LineDiagnosis DifferentialAlveolar cellsCytokeratinKeratinmedicineAnimalsHumansLungchemistry.chemical_classificationTracheal EpitheliumLungrespiratory systemmedicine.diseaseCell Transformation Neoplasticmedicine.anatomical_structurechemistryCell cultureCarcinoma Squamous CellKeratinsAdenocarcinomaCattleVirchows Archiv B Cell Pathology Including Molecular Pathology
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Side-specific effects by cadmium exposure: Apical and basolateral treatment in a coculture model of the blood–air barrier

2010

Cadmium (Cd{sup 2+}) is a widespread environmental pollutant, which is associated with a wide variety of cytotoxic and metabolic effects. Recent studies showed that intoxication with the heavy metal most importantly targets the integrity of the epithelial barrier. In our study, the lung epithelial cell line, NCI H441, was cultured with the endothelial cell line, ISO-HAS-1, as a bilayer on a 24-well HTS-Transwell (registered) filter plate. This coculture model was exposed to various concentrations of CdCl{sub 2}. The transepithelial electrical resistance decreased on the apical side only after treatment with high Cd{sup 2+} concentrations after 48 h. By contrast, a breakdown of TER to less t…

Time FactorsCell SurvivalToxicologyTight JunctionsProinflammatory cytokineAlveolar cellsCadmium ChlorideCell Line TumorElectric ImpedancemedicineHumansViability assayRespiratory systemFragmentation (cell biology)Cell ShapePharmacologyBlood-Air BarrierDose-Response Relationship DrugChemistryCell PolarityEndothelial CellsEpithelial CellsBlood–air barrierAdherens JunctionsMolecular biologyCoculture TechniquesEndothelial stem cellmedicine.anatomical_structureCytoprotectionImmunologyCytokinesCalciumInflammation MediatorsIntracellularToxicology and Applied Pharmacology
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